Excessive gum exposure when smiling is most commonly caused by a hyperactive upper lip elevator muscle. Precise neurotoxin placement reduces the pull and creates a more balanced smile.
get startedA gummy smile refers to the visible exposure of more gum tissue than is conventionally considered aesthetically balanced when smiling. The clinical threshold is typically more than 3–4mm of gum tissue visible above the upper teeth during a full smile, though patient perception and aesthetic preference vary.
It’s a common concern — affecting an estimated 10–15% of the adult population — and one that is highly treatable non-surgically in the majority of cases where muscle hyperactivity is the primary driver.
Patients with a gummy smile often describe years of self-consciousness about their smile — covering their mouth when they laugh, avoiding photos that show their full smile, or feeling that their teeth look small relative to the amount of gum visible. Many are surprised to learn that an injectable treatment can correct it in one appointment without surgery.
A gummy smile has three potential drivers, which may appear independently or in combination.
Hyperactive upper lip elevator muscles: The most common cause. The levator labii superioris alaeque nasi (LLSAN) and related muscles pull the upper lip upward when smiling. In patients with a hyperactive LLSAN, the lip travels higher than average — exposing more gum tissue than intended. This is the component most consistently addressable with neurotoxin.
Short or hyperactive upper lip: Some patients have an anatomically short upper lip or a lip that travels more than the average 6–8mm during a full smile. This amplifies the degree of gum exposure even without skeletal or gingival factors.
Vertical maxillary excess: In some patients, the upper jaw sits lower than ideal relative to the lip, pushing more gum tissue into view regardless of muscle activity. This is a skeletal factor that non-surgical treatment addresses only partially.
Patients with a gummy smile typically notice:
A gummy smile driven by muscle hyperactivity tends to remain relatively stable throughout adulthood — it’s a structural and muscular characteristic rather than a progressive aging change. Some patients report it becoming more noticeable as the upper lip thins slightly with age, reducing the tissue that partially conceals the gum line.
Consistent neurotoxin treatment actually produces a mild cumulative effect over time: the muscle atrophies slightly with repeated relaxation, and some patients find that their dose requirement decreases and their duration extends after several treatment cycles.
The approach depends on the degree of gum exposure and what’s driving it.
At CAMI, gummy smile treatment begins with an assessment of how much of the gum exposure is muscular versus structural. The vast majority of patients presenting with this concern have hyperactivity of the LLSAN as the primary driver — which is the component we address with neurotoxin.
Placement is precise and conservative on first treatment. The LLSAN sits in close proximity to other muscles that control upper lip movement and expression — overdosing affects the natural smile in ways that are immediately noticeable and take several months to resolve. We assess response at the two-week mark and adjust at subsequent treatments based on outcome. Most patients find their ideal dose within one to two treatment cycles.

Care guided by experience, precision, and a deep understanding of natural beauty.
get started